For mind, memory and concentration¹

¹Vitamin C and vitamin E help to protect organs such as the eyes, kidneys, nerves, brain, heart, and blood vessels against free radicals.
Folic acid helps to maintain memory function longer in old age.
Pantothenic acid enhances normal mental performance. .


How neurofelixir® works

Antioxidants for your brain cells

Our brain is an impressive and very active organ, functioning as the control center of our body. You can imagine every cell as a little power plant that produces energy. But the productivity also emits toxic pollutants which we call free radicals. Normally, these are intercepted by our endogenous antioxidants.
However, the ratio of free radicals to antioxidants can become distorted by various factors such as stress, age, illness or environmental pollution. Performance decreases, memory deteriorates, energy and drive are lacking. The brain cells become damaged or die completely.
The micronutrients contained in neurofelixir® have an antioxidant* effect and can intercept the harmful free radicals ** thus protecting the cells from damage ***.

20 micronutrients: It’s all in the mix!

Nature offers numerous side-effect-free substances that influence the function and survival of nerve cells. *** A main component amongst the substances contained in neurofelixir® are polyphenols, also called secondary plant substances.
Secondary plant substances have previously been underappreciated in respect to their health benefits*. Since then, both science and the Federal Office of Consumer Protection and Food Safety have confirmed the antioxidant effect of neurofelixir®*.
For neurofelixir®, valuable polyphenols are extracted from plants and combined with vitamins and amino acids. Natural nutrition serves as a model for this process, as a more versatile supply of vital substances helps the body to use these substances more efficiently.

All in one - The preparation is as easy as this

We recommend to take neurofelixir® before midday after a meal.


✓ One package contains 30 sachets à 12.5 g

✓ Can easily be mixed with liquids

✓ Free shaker with every initial order

neurofelixir® capsules

✓ One package contains 180 capsules

✓ Individual dosage - ideal to begin

✓ High quality capsule case - Free of animal remains and 100% vegan

*The polyphenols contained in this product have an antioxidant effect.
**Vitamin C and vitamin E help protect organs such as the eyes, kidneys, nerves, brain, heart and blood vessels against the effects of free radicals.
***Vitamin B2, vitamin C and vitamin E help protect cells against oxidative stress.


neurofelixir® has positive effects on:

What does neurofelixir® contain?

Green Tea

Green tea has been very popular in Asia for thousands of years. Recent studies show that the health-promoting effects are mainly due to a certain ingredient: EGCG and its antioxidant properties*. In the brain, it reduces the formation of toxic proteins, which destroy the nerve cells.¹ ² In addition, the valuable substance is able to improve concentration and attention from a certain dosage onwards.¹

Per daily dose: 909.1 mg green tea extract / of which 500 mg EGCG

Hop cones

Heavy beer consumption is not recommended. But one very specific natural ingredient is: xanthohumol which is found it the hop blossom. Researchers have discovered that it is able to protect our nerve cells⁴ ⁵ and improve our cognitive performance.⁶

Per daily dose: 300 mg of natural extract of hop cones


The bright orange turmeric root is becoming more and more popular. Also in the West the valuable substance curcumin is used diversely due to its antioxidative and anti-inflammatory effect⁷ as well as it’s pain-relieving⁸ and decontaminating properties⁹ which have been determined. Unfortunately, the human body has difficulties absorbing the valuable substance curcumin so that intake in combination with piperine is recommended.

Per daily dose: 526.3 mg turmeric root extract / of which 500 mg curcuminoide


Incense has a long tradition not only in religious contexts but also in medical terms. The resin of the incense tree was used early in the ancient Indian healing art of Ayurveda to alleviate inflammation and joint pain¹⁰ . According to recent studies, boswellic acid, contained in frankincense extract, also seems to reduce the harmful tau proteins, which are partly responsible for brain damage in old age.¹¹

Per daily dose: 235.3 mg / of which 200 mg boswellic acid

Chinese liquorice root

The liquorice root (Glycyrrhiza glabra) contains several substances that have been used medically for centuries - especially to heal respiratory infections. According to recent studies, the anti-inflammatory effect also benefits the brain: the glycyrrhizic acid contained can neutralise a protein secreted by inflammatory cells in the brain.¹² There are also other mechanisms by which the liquorice root protects our nerve cells¹³ ¹⁴ and improves our memory.¹⁵

Per daily dose: 125 mg

Goji berry

The goji berry is used in many ways in traditional Chinese medicine. Due to the gentle manufacturing process, the valuable natural components as well as the sourly-fruity aroma of the whole goji berry are almost completely preserved.

Per daily dose: 500 mg of goji berry powder


The beetroot, which is originally from the Mediterranean region, is known for its bright colour. Studies show that it is able to strengthen the immune system and prevent oxidative stress. ¹⁸

Per daily dose: 500 mg of beetroot powder


The substance quercetin is mainly found in the outer rings of the onion bulb and is classified with the flavonoids, i.e. the plant dyes. By the way: In order to take the daily dose of the protective and preventive substance¹⁹, one would have to eat 1/2 kg of onion bulbs on a daily basis.

Pro Tagesdosis: 300 mg Quercetin


The active ingredient pterostilbene found in blueberries is here the center of our attention. It is related to the somewhat better known resveratrol, derived from red grapes which, however, is difficult for cells to absorb. Current studies show that pterostilbene is able to purify the cells²⁰ ²¹ and to inhibit inflammatory reactions.

Per daily dose: 150 mg of pterostilbene

Black Pepper

The substance piperine contained in black pepper enhances the positive properties of some plant substances. It ensures that the polyphenols are better absorbed by the body, thus increasing bioavailability. This is especially true for turmeric, which normally reaches cells in only very small proportions.

Per daily dose: 105.3 mg of black pepper fruit extract / 100 mg of which piperine

Further ingredients

Contents per daily portion (12,5 g)

Coenzyme Q10

100 mg


150 mg

Folic acid

1000 μg

Pantothenic acid

10 μg

Vitamin B1

50 mg

Vitamin B6

10 mg

Vitamin B12

1000 μg

Vitamin C

250 mg

Vitamin D

20 μg


Demand can be increased by:

  • Illness
  • Stress
  • Aging
  • Environmental impacts
  • Medication intake
  • Unhealthy lifestyle

* The polyphenols contained in this product ensure the antioxidant effect.
** Black pepper increases the bioavailability of the active ingredients.
¹ Chesser, A.S., et al., Epigallocatechin-3-gallate enhances clearance of phosphorylated tau in primary neurons. Nutr Neurosci, 2016. 19(1): p. 21-31.
² Levin, J., et al., The PROMESA-protocol: progression rate of multiple system atrophy under EGCG supplementation as anti-aggregation-approach. J Neural Transm (Vienna), 2016. 123(4): p. 439-45.
⁴ Chang, Y., et al., Xanthohumol-induced presynaptic reduction of glutamate release in the rat hippocampus. Food Funct, 2016. 7(1): p. 212-26
⁵ Yao, J., et al., Xanthohumol, a polyphenol chalcone present in hops, activating Nrf2 enzymes to confer protection against oxidative damage in PC12 cells. J Agric Food Chem, 2015. 63(5): p. 1521-31.
⁶ Zamzow, D.R., et al., Xanthohumol improved cognitive flexibility in young mice. Behav Brain Res, 2014. 275: p. 1-10.
⁸ Di Pierro F, Rapacioli G, Di Maio EA, Appendino G, Franceschi F, Togni S. Comparative evaluation of the pain-relieving properties of a lecithinized formulation of curcumin (Meriva(®)), nimesulide, and acetaminophen. J Pain Res. 2013a;6:201-5
⁹ Pharmacogn Mag. 2014 Jan-Mar; 10(37): 61–65. Curcumin attenuates neurotoxicity induced by fluoride: An in vivo evidence. Chhavi Sharma, Pooja Suhalka, Piyu Sukhwal, Neha Jaiswal, and Maheep Bhatnagar
¹⁰ Bannuru RR, Osani MC, Al-Eid F, Wang C. Efficacy of curcumin and Boswellia for knee osteoarthritis: Systematic review and meta-analysis. Semin Arthritis Rheum. 2018 Mar 10. pii: S0049-0172(18)30002-7.
¹¹ Fathi, E., et al., The Effects of Alpha Boswellic Acid on Reelin Expression and Tau Phosphorylation in Human Astrocytes. Neuromolecular Med, 2016.
¹² Santoro, M., et al., In-vivo evidence that high mobility group box 1 exerts deleterious effects in the 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine model and Parkinson's disease which can be attenuated by glycyrrhizin. Neurobiol Dis, 2016. 91: p. 59-68.
¹³ Link, P., et al., Extracts of Glycyrrhiza uralensis and isoliquiritigenin counteract amyloid-beta toxicity in Caenorhabditis elegans. Planta Med, 2015. 81(5): p. 357-62.
¹⁴ Kilpatrick, K., et al., Chemical induction of Hsp70 reduces alpha-synuclein aggregation in neuroglioma cells. ACS Chem Biol, 2013. 8(7): p. 1460-8.
¹⁵ Cui, Y.M., et al., Effect of glabridin from Glycyrrhiza glabra on learning and memory in mice. Planta Med, 2008. 74(4): p. 377-80.
¹⁷ Nielsen, S.S., et al., Nicotine from edible Solanaceae and risk of Parkinson disease. Ann Neurol, 2013. 74(3): p. 472-7.
¹⁸ Clifford, T., Howatson, G., West, DJ., Stevenson, EJ. (2015): The Potential Benefits of Red Beetroot Supplementation in Health and Disease. Nutrients, 7(4): 2801-2822.
²¹ Vingtdeux, V., et al., AMP-activated protein kinase signaling activation by resveratrol modulates amyloid-beta peptide metabolism. J Biol Chem, 2010. 285(12): p. 9100-13.
²² Capiralla, H., et al., Resveratrol mitigates lipopolysaccharide- and Abeta-mediated microglial inflammation by inhibiting the TLR4/NF-kappaB/STAT signaling cascade. J Neurochem, 2012. 120(3): p. 461-72.
²³ Shoba, G., et al., Influence of piperine on the pharmacokinetics of curcumin in animals and human volunteers. Planta Med, 1998. 64(4): p. 353-356.

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